Grow Taller With Steroids And Steroid Derivatives, Part II

When the website was first beginning I had written a post listing which steroids have been discussed with the possibility of increasing height or stunting growth. The post Grow Taller Using Steroids , Part I” has been one of the most visited posts and it seems that the effects of steroids have never been answered. Would they stimulate or inhibit height?

The answer is that it really depends on the type of steroids one decides to use. Some steroids have been shown to actually increase the growth rate in children with still open growth plates, and others accelerate the senescence of the growth plates and increase the maturity of the bones.

Something we really should be asking ourselves is this “What is the technical definition of steroids?”

It turns out that the term “steroids” refers to any type of laboratory created synthetic hormone which will have similar effects as the sex hormones aka androgens the males of our species releases in their testicles.

Note: This information is below taken from the Steroids.com website.

The definition of an anabolic steroid as defined currently in the United States under (41)(A) is that “anabolic steroid” means any drug or hormonal substance, chemically and pharmacologically related to testosterone (other than estrogens, progestins, corticosteroids, and dehydroepiandrosterone (7).

What we the general public refers to when we use the word “Steroids” are the anabolic steroids type. If we were to use the technical term for steroid found from the field of Chemistry, we would get something different which would not have the same function for the context of the subject we are talking about.

It seems that steroids are then just the synthetic version of testosterone. There has been many studies which show that for males, the hormone that makes them become “men” in the biological definition, is from the androgen testosterone which is made in the male gonads. When a young adolescent male starts going through puberty, they get both the rush of the female androgen estrogen and the male androgen testosterone. The female gonads, the ovaries also produce progesterone but that it not our talk for this post.

It is testosterone which makes males feel stronger, have bigger, thicker muscles, and have increased sexual drive and sexual stamina.

However, there is another type of hormone that is released by the human endocrine system which have similar effects towards making people feel younger, stronger, and increased sexual drive. That is the growth hormones or hGH which is released by the pituitary gland. This is what we as height increase researchers are looking into.

And for me personally, this is what I would also label as a steroid. If a company is manufacturing synthetic versions of the somatropin, I would also call it a steroid.

In my definition of the word “steroids”, it refer to lab derived versions of the Growth Hormones made by the pituitary gland and the Testosterone made by the male gonads.  

There has been many, many people who claim that steroids is bad for a kid who is still growing. Everyone seems to quote the famous horror stories of stunted growth and shrinking testicles, but from what I have researched, there has not been even 1 study which shows the direct link and harm from steroid usage and really bad negative side effects. If a person can direct me to at least 3 PubMed studies showing negative consequences of using steroids within physically mature males I would greatly appreciate it.

If “steroids” are just testosterone and gonadotropin, then why is it bad for a person?

It is bad if a teenage boy who is already having his body filled with hormones get even more. That is like turning an already horny teenage boy even crazier with sexual frustration with more testosterone. It could turn that teenage male potentially violent from sexual repression.

The thing is that I don’t study the science on how to get bigger muscles and how to increase male sexual performance, or stamina. That is for another researcher. Let’s get back to how steroids affect height.

So if steroids are just growth hormones and testosterone, how would they effect the growth process of a person with open growth plates?

As for height, the synthetic steroids for growth hormones derived from the hypothalamus-pituitary gland region of the brain like Genotropin, Somatropin, Humatrope, would actually increase the growth rate and the final height of people who take it. However, the effects may be very small. When we look at the case of Lionel Messi and how he started to take Growth Hormone injections to prevent him from becoming too small in life. The story about Messi and his height was that when he was a young kid, his parents noticed that he was much shorter and smaller than his peers. From doctors testing him, they had concluded that Messi was short statured due to what is known as Growth Hormone Deficiency. The growth plates in his body had the receptors to accept any GH that was in his system, but there was not enough being produced. If Messi did not take the injection, because his body (pituitary glands and adrenal glands) was not producing either the hGH or the IGF-1, he would have ended up much shorter than his current stature of 5′ 7″ which is still on the short side for professional soccer/futbol players. I wrote a post about the unique situation of Messi in the post “Lionel Messi Using Growth Hormone Therapy To Increase Height And Grow Taller”.

I have shown in previous posts that if a male gets too much testosterone into their system while they are still growing ie in their puberty phase, it will convert aka aromatize into the female androgen estrogen and that is what will speed up the rate at which the chondrocytes in the growth plate are being depleted. From my research, it seems that estrogen has been the real activation key on not just determining when the cartilage in the growth plates will disappear, but also when the stage of puberty will begin. This is why people tell a person who decides to use steroids that they are going to end up with stunted growth. If you get too much testosterone into the system, that just leads to more estrogen which will strip the resting zone of the chondrocytes needed for further longitudinal growth.

Clear Examples:

In this article from the LA Times in 2007 entitled “Estrogen’s history as a growth limiter” we find parents who asked physicians to inject their daughter with estrogen to limit her growth and height so that they can always take care of her.

In the post “Alpha, Above Average Testosterone Males Are More Likely To Be Taller, An Endocrine Explanation” I had shown that males who might have slightly higher rates of testosterone rate in their body than their male peers might end up being taller than them as well as more violent and dominant in behavior. However we must be careful not to get too much of the testosterone.

In the post “People Who Lack Functional Estrogen Receptors Become 7 Feet Tall?” I had referenced at least 2 cases of males who had a genetic trait where they did not have receptors in the growth plate to accept the estrogen in their system, which meant that their growth plate cartilage never completely ossified and they ended up being 6′ 8″-7’0″.

Lastly, I have cited this study many times but I will reference it again. The study “Normal bone growth requires optimal estrogen levels: negative effects of both high and low dose estrogen on the number of growth plate chondrocytes.” shows that if a person has too much or too little estrogen in their system, they are going to have stunted growth. There is an optimal level of estrogen. For most men, we go through with puberty later than females so we have on average an extra 2 years of growth before our plates ossify due to the chondrocytes running out. This suggests that having a little extra testosterone might be actually beneficial for height increase but only up a certain point. However, there are a certain portion of the male population that already has too much testosterone and taking the steroids will only lead to decreased final height.

At this point, there is no way to separate which males in a group will steroids actually stimulate height increase and which males steroids would inhibit height growth.

The point that this post is supposed to make is that there is a link between using steroids and possibly stunted growth.

However if we decided to take the other type of “steroids” (which I have defined) in terms of synthetic recombinant growth hormones aka somatropins like genotropin, humatrope, etc. it would not stunt our growth but actually increase it if one is going through the puberty phase.

In addition, there are steroid derivatives like Anavar and Letrozole which have PubMed studies backing up the theory that these aromatizing inhibiting compounds will increase the growth period and stop the chondrocytes from being used up too quickly making the final height increase.

At this point I don’t know if the human body can ever become fully saturated with too much growth hormone from taking GH injections. From the pathologies of Gigantism and Acromegaly and how they affect the human body, it would seem that the human body might not have a saturation point on at least somatropin uptake.

 

 

3 thoughts on “Grow Taller With Steroids And Steroid Derivatives, Part II

  1. Kumanovo

    If youre plates are open try creatine.it increase dht by 40 %.it will help you grow taller by estrogen inhibiting properties directly at receptors,also dht induce longitudinal Growth independent of hgh

  2. Adam

    People with Open Plates should try CJC 1925 DAC it has a half life of 8-11 days, which means it’ll release gh for those 8 days with just one injection. There was a guy on the GrowTallerForum talking about it. It’ll still keep keep igf-1 elevated for about 28 days after using it. By using an anti-estrogen with this, one can concievably increase growth rate, without plate closure.

    “Subcutaneous administration of CJC-1295 resulted in sustained, dose-dependant increases in GH and IGF-1 levels in healthy adults and was safe and relatively well tolerated, particularly at doses of 30 or 60 microg/kg. There was evidence of a cumulative effect after multiple doses. These data support the potential utility of CJC-1295 as a therapeutic agent.” [1] This shows safety and efficacy of CJC-1295. Up to 11 days of elevated IGF-1 from the increase in GH created with the DAC creating a half life of about a week, according to this study, without adverse reaction.”
    here’s the link: http://anabolicminds.com/forum/igf-1-gh/161028-russianstar-experiences-cjc.html
    “Written by Russianstar, This information is copyrited.

    Firstly lets explain what the peptide CJC-1295 DAC is.

    Molecular Formula: C152H252N44O42

    Molecular Weight: 3368.7

    Sequence of CJC1295 (modified) without DAC:

    H-Tyr-(D)Ala-Asp-Ala-Ile-Phe-Thr-Gln-Ser-Tyr-Arg-Lys-Val-Leu-Ala-

    Gln-Leu-Ser-Ala-Arg-Lys-Leu-Leu-Gln-Asp-Ile-Leu-Ser-Arg-NH2

    CJC-1295 is a GHRH (the 44-amino acid long version) with 15 aminos removed, thus a total of 29 amino acids, and bound to DAC it is also called Drug Affinity Complex, and CJC-1295 is often referred to as GHRH with Drug Affinity Complex, this essentialy lengthens its life span.
    This is how its life is lengthened.
    The modification of growth hormone releasing factor with D-Ala, Gln, Ala, and Leu substitutions at positions 2, 8, 15, and 2 create a much more stable peptide with the substitution at position 2 to prevent DPP-IV cleavage, position 8 to reduce asparagine rearrangement or amide hydrolysis to aspartic acid, position 15 to enhance bioactivity, and position 27 to prevent methionine oxidation. By utilising the Drug Affinity Complex technology to GRF, the peptide selectively binds to circulating albumin after subcutaneous administration, thus prolonging its half-life.
    As you may notice its basicly semorelin with 15 aminos removed. This was because semorelin degrades too rapidly to really make it a viable cost efficient option.
    So they bonded it with an attached 3-maleimidopropionic acid (MPA) unit, which results in binding to albumin after exogenous injection into blood plasma, and creates a far longer half life, this is the DAC, or druf affinity complex i made referance to earlier.
    Now it works by this action as its a GHRH, or growth hormone releasing hormone, In the healthy human body, large amounts of growth hormone are stored in the pituitary. The cells within the pituitary release growth hormone in response to signalling by GHRH (Growth Hormone Releasing Hormone) Then the peptide Ghrelin is (of which GHRPs – Growth Hormone Releasing Peptides – are mimetics), inhibited from releasing these stores by Somatostatin. GHRH and Ghrelin act on different populations of somatotropes (GH releasing cells). GHRP and Ghrelin increase the number of somatotropes releasing GH but not the amount released by each cell.
    GHRH affects both the number of secreting cells and – moreso – the amount they are actualy able to secrete. GHRH and Ghrelin are released in specific patterns that vary depending on what the person involved is doing, or has been doing post-exercise. Now CJC-1295 DAC has been proven to stimulate slow wave sleep, and this is the period of sleep when most of your bodys repairing work takes place on muscles and tissues etc.
    Now most people can and will make GH in their own pituitary gland, but not everyone can release it in the amounts needed, so from a medicinal point CJC-1295 DAC can be very beneficial.

    Now for its benefits to reach full potential, somatostatin needs to be inhibited because we have just seen it inhibits gh release, so by using a compound known as an Acetylcholineesterase inhibitor, now Acetylcholineesterase inhibits acetylcholine, and Acetylcholine inhibits somatostatin, so we want Acetylcholine in abundance, so by using an Acetylcholineesterase inhibitor, we allow acetylcholine to inhibit somatostatin, so increasing the amount of gh released when using CJC-1295DAC. Got it? Ok re read that last passage.
    The best of these Acetylcholineesterase inhibitors and there are lots like..
    Physostigmine
    Neostigmine
    Pyridostigmine
    Ambenonium
    Demarcarium
    Rivastigmine
    Phenanthrene derivatives
    Galantamine
    Piperidines
    Donepezil, also known as E2020
    Tacrine, also known as tetrahydroaminoacridine (THA’)
    Edrophonium
    Huperzine A
    Ladostigil,
    Of the ones ive tried Huperzine A is the very best, and fairly cheap, now by adding ECGCs from green tea, as these ECGCs will transport the huperzine better so finding the receptor it needs the effects are even stronger on inhbiting the acetylcholineesterase.
    Otherwise a really good one i used recently was Horny goat weed, its the only one i know that actualy increases acetylcholine as it inhibits the enzyme acetylcholinesterase.
    Personaly i rate this as my favourite peptide, you get nearly a constant surge in gh especialy if you add in say huperzine A, you get all the benefits of hgh, improved body composition, anabolic effects, injury recovery, improved skin, the full works, and at a very good price, far cheaper than real gh.
    I have found that between 2000mcg -5000mcg a week has given me great results, increased bodymass over a 15 week period of nearly 9lbs, thats not weight gained, thats pure muscle and fat loss, You get all the same benefits of hgh, but you get them constantly, not just in one or 2 surges when you use the injections.
    Now i would use this in fat deposits as i noticed a reduction when i did so quite quickly, and directly into a muscle with this is quite painfull to say the least, so thats a no no from me.
    Expect to see results after just a couple of weeks, and the large amounts of igf-1 released will really add some muscle when incoorporated with a good diet and training regime.

    There are no sides ive seen with cjc just positive ones except for the amazingly strong head rush you get about 20 mins after the injection, this a tell tale sign as to wether or not you have the real thing.
    Remember the pulses are not as big as when you use GHRP-6, but rather its a continuous elevation of gh secretion peaking about 2 hours after the injection and staying elevated for 14 days. I use it 4 times a week in 500mcg doses and find this to be perfect for maintaining muscle when unable to train, say because of injury, and improving skin tone and health.
    3500mcg is a far more anabolic dose and muscle mass will be noticeably improved after 4 weeks, and the skin will start to take on that shredded ultra thin look that gh users get before competitions.
    All the benefits of real GH, less injections, less sides, a lot less money…. need i say more.
    Only one thing i should make mntion of, is that according to dat, it can cause gh bleeding, possibly causing reduced gh sensitivity over time.

    I hope you have found this information helpfull, i feel its one of the very best peptides if not the best.

    Russianstars peptide rating 9/10

    Kind regards RS”

  3. Pingback: Does Using Steroids While Still Growing Lead To Stunted Growth, Shorter Height, And Premature Early Growth Plate Closure | Natural Height Growth

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