LSJL Update 10-12-2016

Here’s the link to the last LSJL update.

It seems like my feet have continued to grow from clamping.


Actually there seems to be rather remarkable growth of the 2nd phalanx/phalange bone.  Right foot is loaded with LSJL and left foot is unloaded.  It’s hard to tell if my big toe has grown anymore.  I’ve changed my clamping strategy rather than trying to specifically clamp the epiphysis of the bone.  I’m clamping a part of the bone close to the epiphysis where I can avoid slippage.  This change in clamping strategy may be the cause of the second phalanx growth too. By focusing more on the force rather than location I notice a rush of blood/fluid flow to regions when I release the clamp this may be a beneficial stimulus towards longitudinal bone growth.

My hands look like they’re growing too but there’s no need for pics all I need to is get an xray of one hand and compare them to the existing ones I have.  I may be growing in height again too but until it’s more definitive it’s better to focus on things that are easier to measure.

The Bone Growth Effects Of Growth Hormone On Rich Piana

There is a rather famous YouTuber and bodybuilder Rich Piana who has very publically admitted to his long term consistent usage of steroids as well as growth hormone (Please note that technically steroids and growth hormones are 2 different types of chemicals but for the average person using common words, we can say that growth hormones are in the “steroids” category.). In a couple of his videos, he has talked about what consistent usage of growth hormone has done to his body over the years.

Here are the changes in his bones which he has claimed.

  • Rich has used growth hormones for 10 years straight
  • His shoe size has gone from a size 12 to a size 15.
  • His head size (skull size??), when measured through the wearing of hats, has gone from 7 3/8ths (fitted cap) to 7 3/4ths or even 7 5/8ths
  • His hands have grown
  • His fingers have grown
  • His wrists have grown – by his claim, his wrist has grown 3/4th of an inch (I am assuming in circumference here)
  • His fist have grown 1/2th of an inch in width

So basically he said that the GH injections have grown every single tissue in the body. Here are a few of his other claims, which he used the word “probably” on…

  • heart has grown (probably)
  • brain has grown (probably)
  • intestines have DEFINITELY grown – The evidence for this claim is that his stomach has grown thicker over the years.

It seems that the growth of his stomach was what caused Piana to stop doing GH.

Overall, there are 3 chemical compounds Piana talked about…

  1. Growth Hormone (somatotrophin)
  2. Insulin
  3. IGF-1 (Insulin like Growth Factor Type 1)

His usage of all 3 types of bodybuilding chemicals has caused his intestines to grow. His claim that these same chemicals causing his heart to grow as well will mostly cause him to die at a younger age, which he has sort of fully accepted.

Some Other Information That Piana Talked About

The Cost of Steroid Cycle

  • To compete on the national level of bodybuilding, you would need to be taking about 18 IU – 20 IU of growth hormone a day. The exact type of growth hormone Piana took was Serostim
  • You can buy this growth hormone from a pharmacy, and a kit of Serostim would cost about $2000/kit.
  • There is 7 bottles in 1 kit. He would use 4 bottles a month.
  • The Serostim is about $400-$600/kit
  • If you got it from a doctor, the serostim is about $8000/mth

The usual first cycle that most bodybuilders do, which is sort of like the gateway steroid to the harder stuff, is TEST and DECA, which is a mild cycle – Dosage: 1 cc a week

For Piana, he claimed that after he went through the first trial of steroids ever when he was younger, he put on 28 lbs in 8 weeks, and 22 of those lbs was muscle.

What We Can Take Away From Rich’s Claims

Most people would have never heard of this guy unless they are really deep into the bodybuilding community. This guy is very loud, and makes grand gestures and likes to show off. That is fine since everyone has their own way of life and they will live it in their own style.

We can however learn quite a bit about the long term effects that growth hormone usage will have. The biggest thing is that his shoe size went up 3 sizes. Did the growth hormone cause the irregular bones in his feet to grow bigger? Or maybe the growth hormone caused the ligaments and tendons that connect the bones to muscles and bone to bone to grow” thus expanding the area and distance between each of the bones in the feet, causing the feet to thus expand?

Let’s note that Rich never talked about him noticing any changes in his height after he started using GH.

When he says that his wrist have gotten wider too, I am not sure whether that is just normal appositional bone growth for males between the ages of 20-30 or the result of the GH.

It is obvious that the expansion of the soft tissues like the  ones found in the intestines that are the most notable, and that side effect of GH was what caused Piana to stop using GH, at least on a consistent regular basis.

LSJL studies 5: LSJL device design

This paper discusses a lateral bone loading device.  It mentions a capacity of 40N which I think won’t be enough for lengthening purposes as since lengthening post growth plate senesence is an abnormal task it probably requires very abnormal stimuli.  It’s interesting to look at the device though.

The study mainly mentioned the technical design of the device and no analysis of the applications.

A Mechatronic Loading Device to Stimulate Bone Growth via a Human Knee

“This paper presents the design of an innovative device that applies dynamic mechanical load to human knee joints. Dynamic loading is employed by applying cyclic and periodic force on a target area. The repeated force loading was considered to be an effective modality for repair and rehabilitation of long bones that are subject to ailments like fractures, osteoporosis, osteoarthritis, etc. The proposed device design builds on the knowledge gained in previous animal and mechanical studies. It employs a modified slider-crank linkage mechanism actuated by a brushless Direct Current (DC) motor and provides uniform and cyclic force.”

Here’s an example of what slider crank linkage looks like:


“The functionality of the device was simulated in a software environment and the structural integrity was analyzed using a finite element method for the prototype construction. The device is controlled by a microcontroller that is programmed to provide the desired loading force at a predetermined frequency and for a specific duration. The device was successfully tested in various experiments for its usability and full functionality. The results reveal that the device works according to the requirements of force magnitude and operational frequency. This device is considered ready to be used for a clinical study to examine whether controlled knee-loading could be an effective regimen for treating the stated bone-related ailments{Hopefully bone length is one of those bone-related ailments unfortunately Ping Zhang’s name is not on this paper and he was always the one more interested in bone length}.”

“When a specific loading force is applied to the epiphyses of the femur and tibia, the trabecular bone tissue, which is characterized by axial stress resistance, resists this force from the opposite direction. This results in deformations in that area. These deformations create a variation of the fluid pressure in the intramedullary cavity. This pressure gradient allows the flow of fluids that carry essential nutrients to the bone cortex initiating osteoblast differentiation and osteogenesis, thus helping in repair and regeneration of the bone tissue. This unique reaction makes this procedure an effective treatment for bone rehabilitation. It helps in reduction of healing time of bone fractures and hastens recovery from bone-related injuries and diseases. The lateral stress application is also less strenuous to the knee bone and reduces the amount of force that needs to be applied to get this result.”


B is the force we’re looking for.  The pressure generated by fluid flow not just on the bone but on the stem cells to initiate chondrogenic differentiation.  The pressure on the intact bone may also allow the creation of cartilage canals to enable that requirement for a neo growth plate.

It’s also interesting to note that in the proposed knee loading device the load the entire lateral area of the epiphysis this may be a way to reduce slippage.

” it was decided that the proposed device should be robust enough to produce different magnitudes of linear force up to a maximum of 40 N”<-Since lengthening is not being considered in this study forces required for lengthening may be higher.


The device doesn’t look wide enough for the knee really.  The dimensions of the device listed are:

Length: 0.3 m
Width: 0.1
Height: 0.2

There are about 39 inches in a meter so about 3.9 inches in width.  I don’t know if that’s enough.

Also the device looks more like this kind of clamp:

Then the other clamps we’ve been using.    Although you’d have to make new pads to actually adjust to knee.  Well actually more like:

But the pipe gets in the way of getting around the knee.  Although I’m not really sure that a pipe clamp is superior to the other clamps.  I’m just pointing out that it’s the clamp that looks most like the design mentioned in the study.

Here’s some more details on the device:


Here’s an actual physical prototype:


LSJL Update 9-27-2016

Here’s the last LSJL update.  Here’s the feet images from the this time which have had the best results out of what I’ve been clamping:


The size increase is not due to flattening of the arches as the arch on the right foot actually looks bigger.  You’ll note that the second and third toe look bigger as well.  My wingpsan has increased from 74.5″ to 74.75″ up from the 72.5″ it was before I started LSJL.  It is very difficult however to take a good wingspan picture.

Part of the trouble with LSJL has been slipping when clamping and a possible solution is that rather than clamping the epiphysis of the bone is to clamp the neck of the bone.


Considering the spillover of the second and third toe growth, I’d say it’s probably more important to generate clamping force than it is to be at the optimal location.  Clamping at the neck of the bone also clamps the muscle as well which result in more fluid flowing into the bone.  Also clamping at the neck of the bone gets closer to the bone marrow and one key conclusion I’ve come to my LSJL research so far is that the cortical bone and the outer periosteum(growth in width is difficult as well as growth in length) inhibit bone growth and inner periosteum and bone marrow stimulate bone growth.  Distraction osteogenesis both gets rid of cortical bone via fracture and stimulates the bone marrow via blood clot.

By continuing to clamp the epiphysis of the bone is likely the reason why my length gains plateaued as whenever I tried to increase the clamping force the clamp would slip off.  By clamping the neck of the bone I can continue to increase the clamping force without having to worry about slippage.  Hopefully, this will allow me to get some leg length increases that I’ll be able to report otherwise I’ll see if I can continue to gain in the feet and that’ll be proof of concept that I can use to gain more resources to establish better clamping technology to gain in the legs.

I bought a new clamp for my fingers as the standard six inch clamp was just too big.

The problem with this is all the holes in the clamp that make clamping uncomfortable.  If neck clamping with the Irwin Quick Grip clamp doesn’t work


I can doing the C-class clamp again but I worry even with clamping the neck of the bone rather than the epiphysis there’ll be too much slipping.

We’ll see what happens in one to two weeks.  And if it doesn’t seem to be working I’ll switch it up.  Considering my foot growth if I don’t observe results in a reasonable time frame then it’s time to switch things up


LSJL Update 9-13-2016

I tried hand clamping but I seemed to plateau with it so I’m back to using the C-class but more intensely than before.  Here is me doing some bones with a C-class clamp.  I’ve been getting some progress with my feet at least but that could be because changes in the feet are more noticeable because my shoes feel more snug.  Here’s the last feet images I took for comparison.  The first image there is actually the before picture.  Also the II phalanxes(toe closest to big toe) seems longer as well which makes sense since I’m clamping close by.

But my feet seemed to go up in size very quickly once I change methodology of using the C-class clamp over the hand clamp.  So if there’s no changes in a week then I will try something different.  Michael thought about using two C-class clamps at once.  Ideally, yes you want to gain height but the feet is where I’m getting results and if my right shoe no longer fits that would be hard to deny proof and I could use that proof to acquire more resources to translate to height increase research.

Since the II toe is growing I’m worried less about a precise clamping location and more about clamping force.  Now it is possible that the feet could be flattening but the big toe is already pretty straight.  Well if I can keep getting results than such minutiae won’t matter.

Here’s pictures of my feet:


The right toe is bigger.  I’m not to the point where I need to go up a size for my right foot but I’m closer.

Here’s some unilaterally swollen feet:  The bones don’t physically look longer.  So I don’t think it’s swelling making my feet appear longer.


Here’s another unilaterally swollen foot:


Here’s another:



Myxedema increases hydrostatic pressure by resulting in increased deposition of connective tissue elements like hyaluronic acid and GAGS(chondroitin).  Maybe there’s a way to use the pathology of this disease to safely increase the deposition of connective tissue to possibly increase height.

Increases in deposition of this elements can result from scar tissue.  Perhaps the separation in limb lengthening surgery can be thought of as a form of scar.  Increases in Fibroblast levels also could increase accumulation of connective tissue.  It is worth it to note that FGFR3 decreases height.  Maybe an interesting possibility is that FGFR3 reduces circulation Fibroblast levels and decreases hydrostatic pressure and results in a height increase that way. Thyroid hormone is thought to increase these connective tissue elements.

Maybe in pregnancy the elevated thyroid causes the bump in the stomach during pregnancy and possibly causes the increase in shoe size and height.  Being pregnant can increase the size and production of the thyroid.

Actors like Feldman who had graves disease was not tall at 5’7″.

Other famous with Graves:

Missy Elliott-F 5’2″

Rodney Dangerfield-M 5’10”

1st President Bush-M 6’2″

Maggie Smith-F 5’5″

According to this paper Graves’ disease–acceleration of linear growth., Grave’s may cause an acceleration of linear growth but I could not find anymore beyond the title.

Body height and weight of patients with childhood onset and adult onset thyrotoxicosis.<-Thyrotoxisis is another name for hyperthyroidism

“The present study has compared body height and weight of thyrotoxic female patients of childhood onset and adult onset. The body height of 141 out of 143 (99%) adult-onset thyrotoxic patients was within the range of mean +/- 2SD for the age-matched general Japanese female population. On the other hand, in 42 patients with childhood-onset thyrotoxicosis, 6 (14%) had their height being greater than the mean + 2SD of general population, and 34 (81%) were taller than the mean value. In 86 patients with siblings, 42 (49%) were at least 2 cm taller than their sisters, and 26 (30%) were more than 2 cm shorter than their sisters. The body weight of 27 out of 42 (68%) patients younger than 20 years was not decreased but was even greater than the mean value for the age-matched general population. The results indicate that excessive thyroid hormone in vivo enhances body height in humans. The increased body weight in some young patients suggests that enhanced dietary intake due to increased appetite in hyperthyroidism has overcome the energy loss with increased metabolism.”

If you look at figure 1 you get quite a interesting figure that shows that of females with adult onset hyperthyroidism tend to be taller than the mean(this is not a longitudinal study so hyperthyroidism could not be a direct measurement of height increase).  I could not excise the figure out of the study you will have to look at it directly.

46, XY pure gonadal dysgenesis: a case with Graves’ disease and exceptionally tall stature.

“Growth was arrested with height remaining at 187 cm after normalization of the thyroid function by treatment with an antithyroid agent, although follow-up to monitor growth was limited to 3 months. In some cases of gonadal dysgenesis, then, Graves’ disease may contribute to an abnormally tall stature.”

So we see that Grave’s disease has an impact on height but that the affect on height is variable sometimes an increase and sometimes a decrease.  Maybe there’s some other variable like FGFR3 levels that influence the effects on height.

This provides more evidence that hydrostatic pressure influences height but perhaps some other stimuli is needed like CNP as increases Fibroblastic stimuli would result in more FGFR3 stimuli in some cases.  CNP would cancel that out.