This study provides evidence that supplementation of Quercetin, Kaempferol, and Rutin may affect height during development but you can’t be sure until it’s tested.  The method by which it induces height growth is by increasing the expansion of the bone lacunae per hypertrophic chondrocyte generating more bang for the buck.

Genetically engineered flavonol enriched tomato fruit modulates chondrogenesis to increase bone length in growing animals.

“Externally visible body and longitudinal bone growth is a result of proliferation of chondrocytes[not necessarily; chondrocyte hypertrophy plays a large role]. In growth disorder, there is delay in the age associated increase in height[not always]. The present study evaluates the effect of extract from transgenic tomato fruit expressing AtMYB12 transcription factor on bone health including longitudinal growth. Constitutive expression of AtMYB12 in tomato led to a significantly enhanced biosynthesis of flavonoids in general and the flavonol biosynthesis in particular. Pre-pubertal ovary intact BALB/c mice received daily oral administration of vehicle and ethanolic extract of wild type (WT-TOM) and transgenic AtMYB12-tomato (MYB12-TOM) fruits for six weeks. Animal fed with MYB12-TOM showed no inflammation in hepatic tissues and normal sinusoidal Kupffer cell morphology. MYB12-TOM extract significantly increased tibial and femoral growth and subsequently improved the bone length as compared to vehicle and WT-TOM. Histomorphometry exhibited significantly wider distal femoral and proximal tibial growth plate, increased number and size of hypertrophic chondrocytes in MYB12-TOM which corroborated with micro-CT and expression of BMP-2 and COL-10, marker genes for hypertrophic cells. We conclude that metabolic reprogramming of tomato by AtMYB12 has the potential to improve longitudinal bone growth thus helping in achievement of greater peak bone mass during adolescence.”

What’s significant is that this improves bone growth in normal individuals.

“the proliferative zone contains replicate chondrocytes arranged in columns parallel to the long axis of the bone”

“the proliferative chondrocytes located farthest from the resting zone stop replicating and enlarge to become hypertrophic chondrocytes which subsequently form bone. these cells also maintain the columnar alignment in the hypertrophic zone.”

“growing female mice fed with extract from MYB12-TOM affected formation, quality and length of the bone. Extract from MYB12-TOM significantly increased the length by interstitial growth of the epiphyseal plate of bones by the expansion of the lacunae in the hypertrophic cells in pre-pubertal stage.”

“genetically engineered transgenic tomatoes (MYB12-TOM) expressing a flavonol specific transcription factor from Arabidopsis, AtMYB1214. The fruits of MYB12-TOM accumulated significantly higher amount of flavonols as compared to wild type tomatoes (WT-TOM).”

“expansion of the lacunae in the hypertrophic cells leads to the increased longitudinal growth”

The genetically altered tomatoes had much higher levels of Quercetin, Kaempferol, and Rutin than normal,

“Extracts from MYB12-TOM increased the femur length significantly by 6.1% (P < 0.05) as compared to WT-TOM. In case of tibia, WT-TOM and MYB12-TOM increased the bone length by 5.54% (P < 0.05) and 9.98% (P < 0.001) as compared to control group respectively. Further, comparisons within experimental groups show that MYB12-TOM increased tibial length significantly by 4.2% (P < 0.05) as compared to WT-TOM group”

So it may be possible that Quercetin, Kaempferol, and Rutin supplements to have a significant impact on height during development.

“transgenic MYB12-TOM group exhibited significantly higher COL10a expression from control group (P < 0.001) and WT-TOM (P < 0.001)”

Okay, so here’s the last images and I have some observations and some tweaks.  It seems that it’s mainly the tip of the finger that’s growing and I do have to clamp the tip of my finger to pull on it.  Whereas it’s harder to see any changes in the rest of the finger which is being pulled.

The right pinky is the loaded finger.  I know it’s hard to see anything but I’m just in the testing stages right now.  If there’s every strong enough evidence I can get x-rays and compare it with earlier x-rays I have.  But I don’t think it’s finger pulling that’s effective but rather the inadvertent LSJL that occurs via me clamping my fingers in order to pull on it.

Hydrostatic pressure is the force created by a fluid at rest by clamping you’re disabling the ability of fluid to move.  Hydrostatic pressure is a consistent chondroinducter.  Chondrocytes are the basis for the growth plate.  I’ve tried clamping fingers with a mechanical clamp but didn’t really get significant results.  Although a clamp can generate more force, using your hands is more precise.

So now I’m going to change my methodology a little bit.

I’m just going to hand clamp the middle of the finger to see what effects it has.  One potential difference between the proximal and medial finger bone is that there’s only one epiphysis in the proximal finger bone.  And fluid may not be at rest but simply flow out the other end.

One potential solution is this:

Clamp one epiphysis with a mechanical clamp and clamp the other epiphysis with a hand clamp.

If you look closely at the first image you can see slightly more swelling at the epiphysis of the proximal finger.  It’ll be interesting to see any swelling that occurs in the middle region.

I don’t expect the first image to serve as proof of any means.  Just experimenting to try to find the right methodology and I think LSJL is finally being narrowed down into what actually would be effective for height increase.  Not focusing so much on generating as much force as possible with a clamp but rather clamping in such a way as to inhibit fluid from leaving the bone to generate hydrostatic pressure.

Mesenchymal condensation is the key to growth plate formation.  And you can’t condense if fluid is flowing every which way.  And growth plate cartilage is avascular.